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mouse antibrdu  (Bioss)


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    Bioss mouse antibrdu
    Mouse Antibrdu, supplied by Bioss, used in various techniques. Bioz Stars score: 91/100, based on 5 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/brdu+(a7)+monoclonal+antibody/BrdU+(A7)+Monoclonal+Antibody/pm32302734-78-4-6
    Average 91 stars, based on 5 article reviews
    mouse antibrdu - by Bioz Stars, 2026-10
    91/100 stars

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    Incubation:

    Article Title: Osthole ameliorates cognitive impairments via augmenting neuronal population in APP / PS1 transgenic mice.
    Article Snippet: Alzheimer’s disease (AD) is a neurodegenerative disorder with notable factors of dysfunction in multiple neurological changes, encompassing neuronal loss in the frontal cortex and hippocampal regions.. Dysfunction of proliferation and self-renewal of neural stem cells (NSCs) was observed in AD patients and animals.. Thereby, mobilizing endogenous neurogenesis by pharmacological agents would provide a promising route for neurodegeneration.

    Article Title: Sodium hyaluronate-CNTF gelatinous particles promote axonal growth, neurogenesis and functional recovery after spinal cord injury.
    Article Snippet: Spinal cord was sagittally sectioned at 10mm with a Leica 1850 cryostat (CM-1850, Leica). .. Spinal cord sections were incubated in the following primary antibodies (Ab) at 4 1C for 48 h: mouse anti-neurofilament (Pan) monoclonal Ab (NF, 1:50, Zymed Laboratories, South San Francisco, CA, USA); rabbit anti-b-tubulin-III protein polyclonal Ab (btubulin-III, 1:100, Sigma); rabbit anti-glial fibrillary acidic protein polyclonal Ab (1:150, Zymed Laboratories); mouse anti-BrdU monoclonal Ab (1:70, Bioss, Woburn, MA, USA); and rabbit anti-doublecortin protein polyclonal Ab (DCX, 1:100, Abcam, Cambridge, MA, USA). .. After washing thrice in 0.01 M phosphate-buffered saline, the sections were incubated with Texas Redconjugated goat anti-mouse Ab (1:100, Jackson, West Grove, PA, USA), Cy2-conjugated goat anti-rabbit Ab (1:100, Jackson), Alexa Fluor 546- conjugated donkey anti-rabbit Ab (1:100, Invitrogen, Carlsbad, CA, USA) or Alexa Fluor 488-conjugated donkey anti-mouse Ab (1:100, Invitrogen) for 8 h at room temperature.



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    mouse anti brdu monoclonal ab  (Bioss)
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    Figure 4 The b-tubulin-III–positive neuron-like cells and the differentiation of endogenous progenitors in the sodium hyaluronate-CNTF group. (a) At 1 month PO in the sodium hyaluronate-CNTF group, many small and process-extending b-tubulin-III–positive neuron-like cells (shown by white arrows) were observed at the lesion edge of the host cord. (b) The b-tubulin-III–positive neuron-like cells were immunoreactive for NeuN (shown by white arrows). (c) At 1 month PO in the sodium hyaluronate-CNTF group, the b-tubulin-III–positive neuron-like cells (shown by white arrows) were detectable in the lesion area. (d) High magnification of the boxed area in panel (c). The b-tubulin-III–positive neuronal fiber (shown by white arrowhead) extended and the b-tubulin-III– positive neuron-like cell seemed to form cell contacts (shown by white arrow). (e–h) At 4 days PO in the sodium hyaluronate-CNTF group, <t>BrdU</t> (red) and DCX (green) double-positive cells were observed at the lesion edge of the host cord, with neuron-like appearance. The cells directed by white arrows in panel (g) are shown in panel (h). (i–l) At 11 days PO in the sodium hyaluronate-CNTF group, BrdU (red) and b-tubulin-III (green) double-positive cells were observed in the lesion area. The cell directed by the white arrow in panel (k) is shown in panel (l). The dotted lines indicate the boundary between the host and lesion area. Scale bars: (a, e–g), 100mm; (b, c and h), 50mm; d, 25 mm; (i–k), 75 mm. A full color version of this figure is available at the Spinal Cord journal online.
    Mouse Anti Brdu Monoclonal Ab, supplied by Bioss, used in various techniques. Bioz Stars score: 91/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    Figure 4 The b-tubulin-III–positive neuron-like cells and the differentiation of endogenous progenitors in the sodium hyaluronate-CNTF group. (a) At 1 month PO in the sodium hyaluronate-CNTF group, many small and process-extending b-tubulin-III–positive neuron-like cells (shown by white arrows) were observed at the lesion edge of the host cord. (b) The b-tubulin-III–positive neuron-like cells were immunoreactive for NeuN (shown by white arrows). (c) At 1 month PO in the sodium hyaluronate-CNTF group, the b-tubulin-III–positive neuron-like cells (shown by white arrows) were detectable in the lesion area. (d) High magnification of the boxed area in panel (c). The b-tubulin-III–positive neuronal fiber (shown by white arrowhead) extended and the b-tubulin-III– positive neuron-like cell seemed to form cell contacts (shown by white arrow). (e–h) At 4 days PO in the sodium hyaluronate-CNTF group, BrdU (red) and DCX (green) double-positive cells were observed at the lesion edge of the host cord, with neuron-like appearance. The cells directed by white arrows in panel (g) are shown in panel (h). (i–l) At 11 days PO in the sodium hyaluronate-CNTF group, BrdU (red) and b-tubulin-III (green) double-positive cells were observed in the lesion area. The cell directed by the white arrow in panel (k) is shown in panel (l). The dotted lines indicate the boundary between the host and lesion area. Scale bars: (a, e–g), 100mm; (b, c and h), 50mm; d, 25 mm; (i–k), 75 mm. A full color version of this figure is available at the Spinal Cord journal online.

    Journal: Spinal cord

    Article Title: Sodium hyaluronate-CNTF gelatinous particles promote axonal growth, neurogenesis and functional recovery after spinal cord injury.

    doi: 10.1038/sc.2014.54

    Figure Lengend Snippet: Figure 4 The b-tubulin-III–positive neuron-like cells and the differentiation of endogenous progenitors in the sodium hyaluronate-CNTF group. (a) At 1 month PO in the sodium hyaluronate-CNTF group, many small and process-extending b-tubulin-III–positive neuron-like cells (shown by white arrows) were observed at the lesion edge of the host cord. (b) The b-tubulin-III–positive neuron-like cells were immunoreactive for NeuN (shown by white arrows). (c) At 1 month PO in the sodium hyaluronate-CNTF group, the b-tubulin-III–positive neuron-like cells (shown by white arrows) were detectable in the lesion area. (d) High magnification of the boxed area in panel (c). The b-tubulin-III–positive neuronal fiber (shown by white arrowhead) extended and the b-tubulin-III– positive neuron-like cell seemed to form cell contacts (shown by white arrow). (e–h) At 4 days PO in the sodium hyaluronate-CNTF group, BrdU (red) and DCX (green) double-positive cells were observed at the lesion edge of the host cord, with neuron-like appearance. The cells directed by white arrows in panel (g) are shown in panel (h). (i–l) At 11 days PO in the sodium hyaluronate-CNTF group, BrdU (red) and b-tubulin-III (green) double-positive cells were observed in the lesion area. The cell directed by the white arrow in panel (k) is shown in panel (l). The dotted lines indicate the boundary between the host and lesion area. Scale bars: (a, e–g), 100mm; (b, c and h), 50mm; d, 25 mm; (i–k), 75 mm. A full color version of this figure is available at the Spinal Cord journal online.

    Article Snippet: Spinal cord sections were incubated in the following primary antibodies (Ab) at 4 1C for 48 h: mouse anti-neurofilament (Pan) monoclonal Ab (NF, 1:50, Zymed Laboratories, South San Francisco, CA, USA); rabbit anti-b-tubulin-III protein polyclonal Ab (btubulin-III, 1:100, Sigma); rabbit anti-glial fibrillary acidic protein polyclonal Ab (1:150, Zymed Laboratories); mouse anti-BrdU monoclonal Ab (1:70, Bioss, Woburn, MA, USA); and rabbit anti-doublecortin protein polyclonal Ab (DCX, 1:100, Abcam, Cambridge, MA, USA).

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